slimming tea protein powder chemicals overdosis do

slimming tea protein powder chemicals overdosis
Candidate Master of Sports Sports Aerobics Department of Physical Training Olympic Reserve sports Aerobics SibGIU
Clinical presentation

Vietnam drug usa overdose

Overdose with steroid-like narcotic prohibited substances development usa as a saporta deversion vietnam thailand aggression is designed for the average bow the brain interacts with society isolating minorities of attractive women for destruction under the guise of eradicating prostitution causes manic syndrome of destruction and sale of organs prostitution oppression of dipolomatic relations and the weakening of the country's economic

Look: rebra high open
Bun bun round
Extra not normal: teres major, vastus lateralis, adductor longus
Shoulders go up triceps biseps big but soft
Walk hips wide open
Looks alike alpha
Low urination, low adore, no food intake
Strange society no food in public, snacks, no coffe tea
Hang out with teens simmilarity look coach
Very loud voice
Bullying strangers
Strange sudden reaction on man look traditional tribe any color

Type: Clinical psychopat maniacal disorder deversia Vietnam drug usa
Structur: babuin king slay
Simmilarity: same walking track always same, agenda dayly, planning overtake sosiety
Musculatur: fatty but shaped as body sculpture
Hormone: high testisteron chemical unknown
Look: steroids over stimulated
Attitude: High agressia Overexcited Act pedofelia
Hang out teens & indians
Brain: intake income simmilarity diplogroup hindic
Voice: loud take attantion simmilarity cocain
Brain: wave low, iq low capasity brain low, memmory low
Fobia:dark,river,forest,claustrophobia,tall freeks,old man
Agressia: white women, teen, kids, asian man
Prefer:Open area, high demand, dominant
Type: mussely, wide walk, soft muscls fatty but sculptural
Small penis,pedofilic attitude,low income look alike army
Algorithm: frigid pimp

Complete biochemistry pathway
Edit
Reactant Metabolite Percent of reactant metabolized Half-life Mechanism
Aspartame Phenylethylamine 50% "Rapid"[8]
Aspartame Aspartic acid (aspartate) 40% "Rapid"[8]
Aspartame Methanol 10% "Rapid"[8]
Methanol Formaldehyde 100% 142 mins[45] Alcohol dehydrogenase
Formaldehyde Formic acid 100% 1 min[45] Catalase-peroxide system, cytochrome P450 mixed-function oxidase system, alcohol fehydrogenase
Phenylethylamine Phenylalanine 100% – Phenylalanine decarboxylation
Phenylalanine Phenylpyruvate (PPA)
Phenylpyruvate Phenyllactate (PLA)
Phenylpyruvate Phenylacetate (PAA) Decarboxylation via Ehrlich pathway
Phenylalanine Acetoacetic acid 50%
Phenylalanine Fumaric acid 50%
Phenyllactate (PLA) Phenyllactic acid Lactate dehydrogenase
Phenylalanine Tyrosine Phenylalanine


Aspartame is a methyl ester of the dipeptide of the natural amino acids L-aspartic acid and L-phenylalanine.[4] Under strongly acidic or alkaline conditions, aspartame may generate methanol by hydrolysis. Under more severe conditions, the peptide bonds are also hydrolyzed, resulting in free amino acids.

Beta-aspartame differs from aspartame based upon which carboxyl group of aspartate binds to the nitrogen of phenylalanine.


Cocaine (from French: coca;ne, from Spanish: coca, ultimately from Quechua: k;ka)[14] is a central nervous system (CNS) stimulant. As an extract it is mainly used recreationally and often illegally for its euphoric effects, but it is Schedule II in the U.S. and recognized for its medical value. It is primarily obtained from the leaves of two Coca species native to South America: Erythroxylum coca and E. novogranatense.[15][16] These medicinal herbs naturally contain cocaine and have a history of use among indigenous American peoples. After extraction from the plant, and further processing into cocaine hydrochloride (powdered cocaine), the drug is administered by being either snorted, applied topically to the mouth, or dissolved and injected into a vein. It can also then be turned into free base form (crack cocaine), in which it can be heated until sublimated and then the vapours can be inhaled.[12] Cocaine stimulates the reward pathway in the brain.[16] Mental effects may include an intense feeling of happiness, sexual arousal, loss of contact with reality, or agitation.[12] Physical effects may include a fast heart rate, sweating, and dilated pupils.[12] High doses can result in high blood pressure or high body temperature.[17] Effects begin within seconds to minutes of use and last between five and ninety minutes.[12] As cocaine also has numbing and blood vessel constriction properties, it is occasionally used during surgery on the throat or inside of the nose to control pain, bleeding, and vocal cord spasm.[18]
Cocaine



Penicillins (P, PCN or PEN) are a group of ;-lactam antibiotics originally obtained from Penicillium moulds, principally P. chrysogenum and P. rubens. Most penicillins in clinical use are synthesised by P. chrysogenum using deep tank fermentation[2] and then purified.[3][4] A number of natural penicillins have been discovered, but only two purified compounds are in clinical use: penicillin G (intramuscular or intravenous use) and penicillin V (given by mouth). Penicillins were among the first medications to be effective against many bacterial infections caused by staphylococci and streptococci. They are still widely used today for different bacterial infections, though many types of bacteria have developed resistance following extensive use.
Penicillin



DD-transpeptidase is mechanistically similar to the proteolytic reactions of the trypsin protein family.[5]
<img  decoding="async" width="501" height="411" class="thumbimage" data-file-width="2151" data-file-h

DD-transpeptidase catalytic mechanism
Crosslinking of peptidyl moieties of adjacent glycan strands is a two-step reaction. The first step involves the cleavage of the D-alanyl-D-alanine bond of a peptide unit precursor acting as carbonyl donor, the release of the carboxyl-terminal D-alanine, and the formation of the acyl-enzyme. The second step involves the breakdown of the acyl-enzyme intermediate and the formation of a new peptide bond between the carbonyl of the D-alanyl moiety and the amino group of another peptide unit.[6]
Most discussion of DD-peptidase mechanisms revolves around the catalysts of proton transfer. During formation of the acyl-enzyme intermediate, a proton must be removed from the active site serine hydroxyl group and one must be added to the amine leaving group. A similar proton movement must be facilitated in deacylation. The identity of the general acid and base catalysts involved in these proton transfers has not yet been elucidated.[7] However, the catalytic triad tyrosine, lysine, and serine, as well as serine, lysine, serine have been proposed.[7]






Alexis milk shake
Dangerous drug intake as slimming tea or protein shake
Simmilarity sportsmen boxer reaction on alcs
Simmilarity deversia vietnam agressia to women and kids teens
Human trafficking pimp

Summary
Protein shake type cocain protein penicillin
Reaction alcs simmilar steroid intake
Agitation
Agressive against girls boys teen alike skinny teen alike
War tipe deversia vietnam tai agressia
Look for help to indians friendly simmilaryty by brain regroups family
Afected man asian , egyptian, arab
Small penis, frigid, no erection
Mussel sudden grows but fatty accidity no power
Sudden scare to man big
Caracter bully to teen and women
Sudden character change to Rape pimp bully
Brane parts mostly off
Sudden torette syndrome
Simmilar clinical psychopathy
Synapses maniacal disorder
Dual personality
Low control
Low capasity memmory
Overstimulatet hogh confidance
Blood titr uncnown
Simmilarity cocain
Simmilarity army purpose
Simmilarity milk protein intelerance asian man intake citorizine catalizator